Use Of Reducing Signs Of Alzheimer s Disease - 1773 Words

The use of Curcumin in reducing signs of Alzheimer’s disease Dementia is a drastic failure of cognitive ability that correlates with shrinkage of the hippocampus; the greater the impairment, the likely the smaller the size of the hippocampus. Alzheimer’s Disease is a neurodegenerative disorder, and a form of senile dementia that is characterized by progressive cognitive and sensory impairment in the form of: having a greater difficulty performing tasks, forgetting material that one just read, memory loss, slowed metabolism and atrophy and loss of awareness. With Alzheimer’s being the most common form of dementia, it is estimated that the disease affects 10% of people by the age of 65 and over 50% of people over the age of 85. This disease†¦show more content†¦Due to this, medical professionals have termed the disease â€Å"early onset Alzheimer’s† and make a suspected diagnosis due to symptoms characteristic of the disease. A diagnosis is only confirmed by brain biopsy after death. Unfortunately, due to th is many answers aren’t found until it is too late. Similarly, as researchers are struggling to find a solution and a cure to Alzheimer’s, the epidemic of people struggling with the disease is rapidly growing. In order to effectively treat and reduce the symptoms of Alzheimer’s, we still need to learn more about b-amyloid buildup and tau protein associated neurofibrillary tangles in the brain, which are characteristic of the disease. Similarly, we need to find if these are linked to APOE4 as those who inherit APOE4 have a greater chance of developing Alzheimer’s disease because is does not break down proteins (Molenda-Figueira, 2015). In order to alleviate the affects of Alzheimer’s disease, such as memory loss and build up of tau proteins, the natural substance Curcumin should be looked at (Hamaguchi, Ono, Yamada, 2010). As an anti-inflammatory relative of the turmeric root, this natural, plant based substance shows promising qualities in reducing the effects of Alzheimer’s in mice. (Ma, Zuo, Yang, Ubeda, Gant, Alaverdyan, Teng, Hu, Chen, Maiti, Teter, Cole, Frautschy, 2012). However, with no replicated studies done on humans, currently, the healing